In addition to VDAC2, BAK has been reported to interact constitutively with MCL1 in the MOM of viable cells (Cuconatiet al, 2003;Nijhawanet al, 2003). VDAC isoformknown as VDAC2functions to literally restrain BAK in the MOM in non-stimulated viable cells. However, death stimuli, including that elicited by truncated BID (tBID)an triggered, pro-apoptotic, BH3-only BCL2 family proteindisplaced […]